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What’s next?’Now we want to use this mechanistic information to discover the macromolecular basis for discover discover small molecules that aging aging program and thus the onset of the proteotoxicity with these diseases by modulating aggregation and disaggregation activities are connected, ‘Kelly states cialis viagra . ‘is the hope that inherent by manipulating the protective mechanism in cells, we find a unit-a single drug that would be for a variety of neurodegenerative diseases where protein aggregation useful useful to neurodegeneration ‘.

The Dillin lab at Salk was interested in investigating the link between cellular aging and the onset of proteotoxicity. Thus, the group has from to determine if known, the aging process in the screw by means of RNA interference , a naturally occurring process of suppressing specific gene activity in living able able be slowed down to reduce the activity of the IGF -pathway. In fact, this approach worked and the researchers discovered, when aging was delayed in the onset of proteotoxicity was also moved. In and out,’In turning off this road in and out, we also found that we altered the high molecular weight aggregates-the plaque buildup in the animals,’says Dillin.

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