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From Wyeth Pharmaceuticals.

Therapeutic agents that are the target cytokine system in the brain and to prevent these to prevent these adverse effects of beta-amyloid identified. This line of investigation suggests that agents that could remove the cytokines to improve cognitive function in Alzheimer ‘s disease. Warren Hirst, from Wyeth Pharmaceuticals, Princeton, is an overview of two possible therapeutic strategies. The first is the discovery and development of novel potent, selective and brain-penetrant small molecule beta-secretase inhibitors.

If, however, a new concept emerging that suggests flavonoids do not simply act as antioxidants but exert their biological effects through other mechanisms. A small number of recent studies carried out in models of Alzheimer’s disease have found that oral administration of green tea flavonoids or grape flavonoids reduces in some cases in some cases improved cognition. Dr Williams and colleagues have their own cellular studies on flavonoid called epicatechin that, abundant in a range of foods is ‘We have found that epicatechin protects brain cells from damage by a mechanism independent of its antioxidant activity and shown in laboratory experiments that it will also reduce some aspects of Alzheimer’s disease.

The research group of Ourania M.

The discovery of how the X protein influences liver cell behavior could be used as a cancer therapy, turning the X protein ‘s presence in the liver of patients benefit. Instead of chemotherapy affect influence the X protein ‘s behavior is an alternative cancer therapy, said Andrisani, a professor of basic medical sciences at Purdue School of Veterinary Medicine. Instead of giving a patient chemicals that damage every cell in the body, potential therapeutic on the X protein could target only cancer cells and slows their growth. Research, In this way,ni with first author Wen Horng Wang and G RALd Gregori and Ronald L.

While a lot more research needs to be done, Andrisani team could could work with the X gene someday treatments treatments and the management of gene effects with drugs viable option. .. Andrisani compares the X gene to a driver downhill who do not use the brakes so that the vehicle accelerate to breakneck speed. But before with the X – with the X – gene may proliferating unhealthy at such rate, it may be possible to Andrisanir with drugs counteract. Andrisani said that it was is currently in the power of medicine treatments treatments that precancerous X-expressing cells, can contrive to die on the basis of of their increased tendency. Researchers at other institutions are optimistic about this aspect of the work impact. – In fact, some drugs already exist, may help slow the effects of the X protein in the liver of cancer patients, Spiro Hiotis, surgeon said at New York University who is While a the study.